Bibliographic citations
Díaz, J., (2005). Estado periodontal en pacientes con diabetes mellitus tipo 1 moderadamente controlados y pacientes no diabéticos de 10 a 18 años [Tesis, Universidad Nacional Mayor de San Marcos]. https://hdl.handle.net/20.500.12672/1736
Díaz, J., Estado periodontal en pacientes con diabetes mellitus tipo 1 moderadamente controlados y pacientes no diabéticos de 10 a 18 años [Tesis]. PE: Universidad Nacional Mayor de San Marcos; 2005. https://hdl.handle.net/20.500.12672/1736
@misc{renati/489737,
title = "Estado periodontal en pacientes con diabetes mellitus tipo 1 moderadamente controlados y pacientes no diabéticos de 10 a 18 años",
author = "Díaz Suyo, Juan Antonio",
publisher = "Universidad Nacional Mayor de San Marcos",
year = "2005"
}
In this study were compared periodontal status between 26 subjects with Type 1 Diabetes Mellitus and 52 non diabetic subjects, 10-18 years old age ranged. To evaluate periodontal status of both, diabetics and non diabetics subjects, were used periodontal parameters such as: gingival index, plaque index, hard deposits index, probing depth and loss of attachment. Gingival index was in diabetic patients 1.18±0.45 and in non diabetics patients 1.12±0.42 (p>0.05). Probing depth was in diabetic patients 2.25±0.36 and in non diabetic patients 2.31±0.26 (p> 0.05). Loss of attachment was in diabetics patients 0.07±0.15 and in non diabetic patients 0.04±0.06 (p>0.05). Hard deposit index was in diabetic patients 0.62±0.68 and in non diabetic patients 0.49±0.66 (p>0.05). Plaque index was in diabetic patients 2.42±0.40 and in non diabetic patients 2.31±0.52 (p>0.05). This periodontal parameters did not show significant differences between both diabetic and non diabetic patients, neither when compared by sex, perhaps by the fact that diabetic patients were marginally controlled. It may support the hypothesis that there are differences in susceptibility to periodontal disease among populations of Type 1 Diabetes Mellitus patients, with subpopulations of varying sizes having increased disease susceptibility that could have been determined by genetic factors.
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